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p-p65 rabbit ab antibody  (Cell Signaling Technology Inc)


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    Structured Review

    Cell Signaling Technology Inc p-p65 rabbit ab antibody
    Antibody information used in the article.
    P P65 Rabbit Ab Antibody, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/p-p65+rabbit+ab+antibody/anti+p38/pmc09574058-4-0-6
    Average 90 stars, based on 1 article reviews
    p-p65 rabbit ab antibody - by Bioz Stars, 2026-09
    90/100 stars

    Images

    1) Product Images from "Ningmitai capsules have anti-inflammatory and pain-relieving effects in the chronic prostatitis/chronic pelvic pain syndrome mouse model through systemic immunity"

    Article Title: Ningmitai capsules have anti-inflammatory and pain-relieving effects in the chronic prostatitis/chronic pelvic pain syndrome mouse model through systemic immunity

    Journal: Frontiers in Pharmacology

    doi: 10.3389/fphar.2022.949316

    Antibody information used in the article.
    Figure Legend Snippet: Antibody information used in the article.

    Techniques Used:

    Decreased prostatic inflammation and inflammatory pain-related pathway activation in mice. (A) Sections of prostate tissues were stained with HE. Scale bar = 50 μm; CTL = control; EAP = experimental autoimmune prostatitis mice; NMT: Ningmitai-treated mice. (B) Inflammatory scores of the CTL, EAP, and NMT groups. n = 6. (C) Thirteen inflammatory-related factors in the CTL, EAP, and NMT groups. n = 6. (D) Western blotting analyzing of p-JNK, JNK, p-ERK, ERK, p-P38, P38, p-P65, P65, p-STAT3, STAT3, and GAPDH expressions as well as a statistical analysis of the results in the CTL, EAP, and NMT groups. n = 6. (*: p < 0.05; **: p < 0.01)
    Figure Legend Snippet: Decreased prostatic inflammation and inflammatory pain-related pathway activation in mice. (A) Sections of prostate tissues were stained with HE. Scale bar = 50 μm; CTL = control; EAP = experimental autoimmune prostatitis mice; NMT: Ningmitai-treated mice. (B) Inflammatory scores of the CTL, EAP, and NMT groups. n = 6. (C) Thirteen inflammatory-related factors in the CTL, EAP, and NMT groups. n = 6. (D) Western blotting analyzing of p-JNK, JNK, p-ERK, ERK, p-P38, P38, p-P65, P65, p-STAT3, STAT3, and GAPDH expressions as well as a statistical analysis of the results in the CTL, EAP, and NMT groups. n = 6. (*: p < 0.05; **: p < 0.01)

    Techniques Used: Activation Assay, Staining, Control, Western Blot



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    Cell Signaling Technology Inc rabbit polyclonal anti phospho nf kb p65 ser536 p p65 abs
    FIGURE 5 | Activation of p38, p44/42 MAPK and <t>NF-kB</t> <t>p65</t> in DC infected with wild type USA300 or esx mutants. The activation of intracellular pathway involving p38, p42/44 MAPK, and NF-kB <t>p65</t> <t>subunit</t> was investigated by western blotting analysis of the phosphorylated isoforms in whole cell extracts prepared from DC infected for 1 (A) and 3 h (B) with wild type USA300 or esx mutants. β-actin levels were analyzed as control for protein loading. Fold changes of each analyzed protein, indicated at the bottom of each immunoblot, were calculated by dividing values obtained in infected conditions with those of the uninfected counterpart. A representative experiment, out of 3 independent experiments performed that yielded similar results, is shown.
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    Image Search Results


    The mRNA expression of ITGAV, FAK, PLC, PKC, p65, ERK, JNK, p38, PI3K, Akt, Bax, Bcl2 , and Caspase 3 in E.tenella host cells.

    Journal: Poultry Science

    Article Title: Pathogenic mechanism of Eimeria tenella Et MIC2 promotes Eimeria tenella invasion and inhibits host cell apoptosis through binding to the ITGAV receptor

    doi: 10.1016/j.psj.2026.106922

    Figure Lengend Snippet: The mRNA expression of ITGAV, FAK, PLC, PKC, p65, ERK, JNK, p38, PI3K, Akt, Bax, Bcl2 , and Caspase 3 in E.tenella host cells.

    Article Snippet: p-p65 Rabbit Ab , Bioss , bs-0982R , 1: 1500.

    Techniques: Expressing

    The protein activity changes of ITGAV, FAK, PLC, PKC, p65, ERK, JNK, p38, PI3K, Akt, Bax, Bcl2, and Caspase 3 in E.tenella host cells.

    Journal: Poultry Science

    Article Title: Pathogenic mechanism of Eimeria tenella Et MIC2 promotes Eimeria tenella invasion and inhibits host cell apoptosis through binding to the ITGAV receptor

    doi: 10.1016/j.psj.2026.106922

    Figure Lengend Snippet: The protein activity changes of ITGAV, FAK, PLC, PKC, p65, ERK, JNK, p38, PI3K, Akt, Bax, Bcl2, and Caspase 3 in E.tenella host cells.

    Article Snippet: p-p65 Rabbit Ab , Bioss , bs-0982R , 1: 1500.

    Techniques: Activity Assay

    Antibody information used in the article.

    Journal: Frontiers in Pharmacology

    Article Title: Ningmitai capsules have anti-inflammatory and pain-relieving effects in the chronic prostatitis/chronic pelvic pain syndrome mouse model through systemic immunity

    doi: 10.3389/fphar.2022.949316

    Figure Lengend Snippet: Antibody information used in the article.

    Article Snippet: p-P65 Rabbit Ab , 1:1,000(WB) , CST (3,031).

    Techniques:

    Decreased prostatic inflammation and inflammatory pain-related pathway activation in mice. (A) Sections of prostate tissues were stained with HE. Scale bar = 50 μm; CTL = control; EAP = experimental autoimmune prostatitis mice; NMT: Ningmitai-treated mice. (B) Inflammatory scores of the CTL, EAP, and NMT groups. n = 6. (C) Thirteen inflammatory-related factors in the CTL, EAP, and NMT groups. n = 6. (D) Western blotting analyzing of p-JNK, JNK, p-ERK, ERK, p-P38, P38, p-P65, P65, p-STAT3, STAT3, and GAPDH expressions as well as a statistical analysis of the results in the CTL, EAP, and NMT groups. n = 6. (*: p < 0.05; **: p < 0.01)

    Journal: Frontiers in Pharmacology

    Article Title: Ningmitai capsules have anti-inflammatory and pain-relieving effects in the chronic prostatitis/chronic pelvic pain syndrome mouse model through systemic immunity

    doi: 10.3389/fphar.2022.949316

    Figure Lengend Snippet: Decreased prostatic inflammation and inflammatory pain-related pathway activation in mice. (A) Sections of prostate tissues were stained with HE. Scale bar = 50 μm; CTL = control; EAP = experimental autoimmune prostatitis mice; NMT: Ningmitai-treated mice. (B) Inflammatory scores of the CTL, EAP, and NMT groups. n = 6. (C) Thirteen inflammatory-related factors in the CTL, EAP, and NMT groups. n = 6. (D) Western blotting analyzing of p-JNK, JNK, p-ERK, ERK, p-P38, P38, p-P65, P65, p-STAT3, STAT3, and GAPDH expressions as well as a statistical analysis of the results in the CTL, EAP, and NMT groups. n = 6. (*: p < 0.05; **: p < 0.01)

    Article Snippet: p-P65 Rabbit Ab , 1:1,000(WB) , CST (3,031).

    Techniques: Activation Assay, Staining, Control, Western Blot

    FIGURE 5 | Activation of p38, p44/42 MAPK and NF-kB p65 in DC infected with wild type USA300 or esx mutants. The activation of intracellular pathway involving p38, p42/44 MAPK, and NF-kB p65 subunit was investigated by western blotting analysis of the phosphorylated isoforms in whole cell extracts prepared from DC infected for 1 (A) and 3 h (B) with wild type USA300 or esx mutants. β-actin levels were analyzed as control for protein loading. Fold changes of each analyzed protein, indicated at the bottom of each immunoblot, were calculated by dividing values obtained in infected conditions with those of the uninfected counterpart. A representative experiment, out of 3 independent experiments performed that yielded similar results, is shown.

    Journal: Frontiers in cellular and infection microbiology

    Article Title: Staphylococcus aureus Esx Factors Control Human Dendritic Cell Functions Conditioning Th1/Th17 Response.

    doi: 10.3389/fcimb.2017.00330

    Figure Lengend Snippet: FIGURE 5 | Activation of p38, p44/42 MAPK and NF-kB p65 in DC infected with wild type USA300 or esx mutants. The activation of intracellular pathway involving p38, p42/44 MAPK, and NF-kB p65 subunit was investigated by western blotting analysis of the phosphorylated isoforms in whole cell extracts prepared from DC infected for 1 (A) and 3 h (B) with wild type USA300 or esx mutants. β-actin levels were analyzed as control for protein loading. Fold changes of each analyzed protein, indicated at the bottom of each immunoblot, were calculated by dividing values obtained in infected conditions with those of the uninfected counterpart. A representative experiment, out of 3 independent experiments performed that yielded similar results, is shown.

    Article Snippet: Blots were incubated with rabbit polyclonal antiphospho-p38 MAPK (Thr180/Tyr182) (p-p38), rabbit polyclonal anti-phospho-p44/42 MAPK (Thr202/Tyr204) (p-p44/42) and rabbit polyclonal anti-phospho-NF-kB p65 (Ser536) (p-p65) Abs (Cell Signaling Technology, Danvers, MA).

    Techniques: Activation Assay, Infection, Western Blot, Control